The hypothesis that IL-10 induced by saliva is involved with disease progression during infection is supported by a substantial enhancement in lesion development and parasitic burden in mice which were co-inoculated with saliva and parasites
The hypothesis that IL-10 induced by saliva is involved with disease progression during infection is supported by a substantial enhancement in lesion development and parasitic b
Proc
Proc. injury or inflammation. Chronic ongoing extracellular SK1-IN-1 release of cytotoxic enzymes permanently damages host tissues, playing a pivotal role in the pathogenesis of